DcR3: From Driver of Tumor Immune Evasion to Inflammation Brake—Translational Prospects of a Two-Faced Target
DcR3 promotes immune escape in the tumor microenvironment while acting as inhibitory feedback in chronic inflammation. This article reviews its bidirectional mechanisms, sources of expression regulation, variable-control essentials on both research f
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Keywords: DcR3,Tumor immune evasion,Chronic inflammation,Immunosuppression,Biomarker,UA019076
Note: The full article is published in Chinese. Please refer to the Chinese version for complete experimental details, figures and product references. For product inquiries, visit www.ua-bio.com.
This article is reviewed and published by the technical expert team of UA
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